LoginNodeless superconducting gap and electron-boson coupling in (La,Pr,Sm)3Ni2O7 films
21 May 2026 at 14:00
Science, Ahead of Print.
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Received — 27 May 2026
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Journals
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AAAS: Table of Contents
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Nodeless superconducting gap and electron-boson coupling in (La,Pr,Sm)3Ni2O7 films
Science, Ahead of Print.
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Cell
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Respiratory viral infections prime accelerated lung cancer growth
Severe COVID-19 is associated with an increased subsequent risk of lung cancer. Viral pneumonia induces durable lung epigenetic imprinting that promotes tumor-supportive neutrophils and impairs T cell immunity, which is reversible with combined CXCR2 inhibition and PD-L1 blockade.
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Respiratory viral infections prime accelerated lung cancer growth
11 March 2026 at 08:00
Severe COVID-19 is associated with an increased subsequent risk of lung cancer. Viral pneumonia induces durable lung epigenetic imprinting that promotes tumor-supportive neutrophils and impairs T cell immunity, which is reversible with combined CXCR2 inhibition and PD-L1 blockade.
Received — 4 April 2026
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Journals
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Ferritin aggregation cell engager for CAR T avidity engineering against refractory leukemias
Li et al. developed a ferritin aggregation cell engager that helps CAR T cells better recognize and attack leukemia cells without re-engineering the CAR itself. This versatile platform overcomes antigen modulation and enables combination with chemotherapy.
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Ferritin aggregation cell engager for CAR T avidity engineering against refractory leukemias
9 March 2026 at 08:00
Li et al. developed a ferritin aggregation cell engager that helps CAR T cells better recognize and attack leukemia cells without re-engineering the CAR itself. This versatile platform overcomes antigen modulation and enables combination with chemotherapy.
Received — 14 March 2026
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Journals
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Cell Death Discovery nature.com science feeds
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MAPK14/SLC7A11/GPX4 axis dysregulation drives podocyte ferroptosis via mediating glycerophospholipid metabolism
Cell Death Discovery, Published online: 11 March 2026; doi:10.1038/s41420-026-02990-7MAPK14/SLC7A11/GPX4 axis dysregulation drives podocyte ferroptosis via mediating glycerophospholipid metabolism
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MAPK14/SLC7A11/GPX4 axis dysregulation drives podocyte ferroptosis via mediating glycerophospholipid metabolism
11 March 2026 at 08:00
Cell Death Discovery, Published online: 11 March 2026; doi:10.1038/s41420-026-02990-7
MAPK14/SLC7A11/GPX4 axis dysregulation drives podocyte ferroptosis via mediating glycerophospholipid metabolism