Normal view
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cs.AI, q-bio.NC updates on arXiv.org
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TrafficMoE: Heterogeneity-aware Mixture of Experts for Encrypted Traffic Classification
arXiv:2603.29520v1 Announce Type: cross Abstract: Encrypted traffic classification is a critical task for network security. While deep learning has advanced this field, the occlusion of payload semantics by encryption severely challenges standard modeling approaches. Most existing frameworks rely on static and homogeneous pipelines that apply uniform parameter sharing and static fusion strategies across all inputs. This one-size-fits-all static design is inherently flawed: by forcing structured
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cs.AI, q-bio.NC updates on arXiv.org
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Mean Masked Autoencoder with Flow-Mixing for Encrypted Traffic Classification
arXiv:2603.29537v1 Announce Type: cross Abstract: Network traffic classification using self-supervised pre-training models based on Masked Autoencoders (MAE) has demonstrated a huge potential. However, existing methods are confined to isolated byte-level reconstruction of individual flows, lacking adequate perception of the multi-granularity contextual relationship in traffic. To address this limitation, we propose Mean MAE (MMAE), a teacher-student MAE paradigm with flow mixing strategy for bu
Mean Masked Autoencoder with Flow-Mixing for Encrypted Traffic Classification
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cs.AI, q-bio.NC updates on arXiv.org
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Building evidence-based knowledge graphs from full-text literature for disease-specific biomedical reasoning
arXiv:2603.28325v2 Announce Type: replace-cross Abstract: Biomedical knowledge resources often either preserve evidence as unstructured text or compress it into flat triples that omit study design, provenance, and quantitative support. Here we present EvidenceNet, a framework and dataset for building disease-specific knowledge graphs from full-text biomedical literature. EvidenceNet uses a large language model (LLM)-assisted pipeline to extract experimentally grounded findings as structured evi
Building evidence-based knowledge graphs from full-text literature for disease-specific biomedical reasoning
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(Multiomics OR Omics) AND (Pancreatic)
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Gut-Brain Axis Dysregulation in Inflammatory Bowel Disease: Implications for Coagulation Abnormalities and Extraintestinal Manifestations
Int J Gen Med. 2026 Mar 24;19:590621. doi: 10.2147/IJGM.S590621. eCollection 2026.ABSTRACTInflammatory bowel disease (IBD) involves chronic intestinal inflammation driven by gut-brain axis imbalance, fostering complications through an "inflammation-neuro-coagulation" triad. Current staging systems inadequately capture the dynamics of this multidimensional network. Therefore, integrated multi-omics analyses-including metagenomics, metabolomics, and single-cell transcriptomics-are essential to con
Gut-Brain Axis Dysregulation in Inflammatory Bowel Disease: Implications for Coagulation Abnormalities and Extraintestinal Manifestations
Int J Gen Med. 2026 Mar 24;19:590621. doi: 10.2147/IJGM.S590621. eCollection 2026.
ABSTRACT
Inflammatory bowel disease (IBD) involves chronic intestinal inflammation driven by gut-brain axis imbalance, fostering complications through an "inflammation-neuro-coagulation" triad. Current staging systems inadequately capture the dynamics of this multidimensional network. Therefore, integrated multi-omics analyses-including metagenomics, metabolomics, and single-cell transcriptomics-are essential to construct dynamic models that monitor coagulation, microbiome, and metabolism for precise assessment of disease activity and thrombotic or bleeding risks. Interventions targeting gut-brain axis nodes, such as eliminating tissue factor-positive (TFβΊ) T cells or modulating vagal activity, show potential to disrupt the inflammation-coagulation cycle, although rigorous randomized trials are still needed. Artificial intelligence (AI)-assisted systems that integrate real-time biomarker monitoring with multi-omics predictions represent a novel paradigm for managing IBD-related coagulation dysfunction. Key challenges include elucidating gut-brain-liver axis regulation of coagulation and characterizing platelet functional heterogeneity. Future efforts must prioritize ethically compliant multi-omics platforms and racially stratified risk models to advance personalized coagulation management in IBD.
PMID:41913906 | PMC:PMC13033200 | DOI:10.2147/IJGM.S590621