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DNA damage burden causes selective CUX2 neuron loss in neuroinflammation

βœ‡Nature - Issue - nature.com science feeds
By: Laura Morcom Β· Wenlong Xia Β· Zhaoyang Xu Β· Yashika Awasthi Β· Celine Geywitz Β· Matthew O. Ellis Β· Tomas Noli Β· Amel Zulji Β· Daniel Yamamoto Β· Gemma C. Girdler Β· Li Kai Β· Keying Zhu Β· Mingming Wei Β· Xiao-Yan Tang Β· Kimberly K. Hoi Β· Julio Gonzalez-Maya Β· Greg J. Duncan Β· Adrien M. Vaquie Β· Diana Gold Diaz Β· Riki Kawaguchi Β· Erdong Liu Β· Yu Sun Β· Denny Yang Β· Gregory D. Jordan Β· I-Ling Lu Β· Staffan Holmqvist Β· Theresa Bartels Β· Katherine Ridley Β· Jennifer Ja-Yoon Choi Β· Santos J. Franco Β· Eric J. Huang Β· Ben Emery Β· Daniel Geschwind Β· Lucas Schirmer Β· Gabriel Balmus Β· Brian Popko Β· Stephen P. J. Fancy Β· David H. Rowitch

Nature, Published online: 01 April 2026; doi:10.1038/s41586-026-10310-3

DNA damage burden and inadequate repair in CUX2+ cortical layer 2/3 excitatory neurons contributes to selective vulnerability in neuroinflammatory injury.
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tRF-3005a regulates exon skipping of SPAG4 by interacting with RALY to drive gastric cancer progression

βœ‡Cell Death Discovery nature.com science feeds
By: Huaiping Cui Β· Yancong Yuan Β· Yizhe Yin Β· Ruihong Gao Β· Zhaodong Liu Β· Lipan Peng Β· Jinshen Wang Β· Zhu Wang Β· Tingting Song Β· Jinglei Liu

Cell Death Discovery, Published online: 24 March 2026; doi:10.1038/s41420-026-03049-3

tRF-3005a regulates exon skipping of SPAG4 by interacting with RALY to drive gastric cancer progression
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